Frederic Veyrier

Academic title(s): 

Associate Professor, INRS-Institut Armand-Frappier

Frederic Veyrier
Contact Information
Address: 

531 Boul. des Prairies

Laval (Québec) H7V1B7

Canada

Phone: 
450) 687 5010 # 8831
Email address: 
frederic.veyrier [at] iaf.inrs.ca
Division: 
Adjunct Members
Branch: 
Microbiology
Graduate supervision: 

ACCEPTING GRADUATE STUDENTS

Biography: 

Frédéric Veyrier completed his doctoral studies at McGill University, Montreal (Qc) on the evolution of the human pathogen Mycobacterium tuberculosis. He returned to France in 2010 as a postdoctoral researcher at the Institut Pasteur, Paris. His research then focused on the common evolution of nasopharyngeal pathogens such as Neisseria meningitidis, Haemophilus influenzae or Moraxella catarrhalis. Specifically, he studied how bacterial metal transport and the cell envelope evolved to adapt to the harsh and competitive environment of the nasopharynx.

Since 2015, he has been a professor at the INRS-Centre Armand-Frappier Santé-Biotechnologie in Canada, and a FRQS research fellow (J1 and J2), pursuing his work on understanding the mechanisms and consequences of the evolution of human bacterial symbionts and more particularly on the determinants of host or tissue tropism.

Awards, honours, and fellowships: 

2021/7 - 2025/7 Research Scholars - Junior 2 - 295,451 (Canadian dollar)

Fonds de recherche du Québec - Santé (FRQS)

2017/7 - 2021/7 Research Scholars - Junior 1 - 280,000 (Canadian dollar)

Fonds de recherche du Québec - Santé (FRQS)

Current research: 

My current research program aims at understanding bacterial human symbiosis (fundamental research) to ultimately find new avenues for diagnostic and treatments (applied research). Our specificity is to look for stepwise ancestral events at different nodes of evolution (including also the ones not directly linked to pathogens speciation) that have drastic consequences on the pathogens as we know them today (“butterfly effect”) and understand what could have been their contribution to the ecological niche adaptation, enhanced colonization and/or virulence of the pathogenic species. This could be termed the intra-genus/family evolution. For this, we study different trajectory of evolution using Mycobacteria, Neisseria, Leptospira and Acinetobacter genera as models.

Selected publications: 
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